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The proteome of cblC defect: in vivo elucidation of altered cellular pathways in humans
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Value:50MG
Research indicates that, over the loss of critical transducer molecules within cell, such as MAPKs, allows adaptor proteins of receptors to interact with other proteins, which mobilize certain cell death mechanisms depending on the presence or absence of p62 (Goodall et al., 2016)
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