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Originally identified in the 1970s during investigations into sleep-associated neuropeptides, DSIP has since been examined across a broad range of experimental models involving hypothalamic-pituitary signaling, biological rhythm regulation, and peptide-mediated cellular communication [1]
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If cells that meet the conditions for pyroptosis also exhibit elevated ROS, do they concurrently undergo apoptosis?” based on prior findings, we consider these conjectures to be theoretically valid
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