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Alopecia associated with dimethyl fumarate treatment for multiple sclerosis | Wiener Medizinische Wochenschrift | Springer Nature Link
This, in turn, causes hyperactivation of the MEK-ERK pathway and programmed cell death of neurons [28]
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depletion of LSD1 leads to increased LPAR6 expression, which promotes paxillin phosphorylation and enhances migration and invasion of androgen-independent PCa cells ( These findings collectively depict continuous LPA metabolic pathway activation during PCa progression, particularly during transition to invasive phenotypes
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