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MOTS-c treatment resulted in a significant upregulation of genes related to mitochondrial biogenesis ( PGC-1α, NRF1 ), mitochondria-encoded genes ( MT-ND1, MT-CO1 ) and OXPHOS-related genes, leading to the biogenesis of more mitochondria with abundant cristae, which is associated with increased respiratory capacity [44, 80, 81]
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