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Upon nicotine treatment, phosphorylation of AKT and ERK is stimulated, enhancing cancer cell proliferation, and this effect is significantly reversed by Ξ±7nAChR antagonists such as Ξ±-bungarotoxin [115]
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The peptide's mobilization of stem cell populationsβincluding dermal stem cells and endothelial progenitor cellsβsupports tissue renewal through recruitment of fresh cellular populations capable of robust synthetic activity
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