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The urgency of the issue cannot be underestimated, as precious time and resources that could be allocated much more constructively and usefully in this time of serious need are being poured into approaches that offer little to no hope of prevention

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Overexpression of TFAM in astrocytes induces mtDNA protection against Aβ1-42 peptide, ultimately protecting neurons (101), while tissue-specific or partial deletion of the TFAM protein leads to respiratory chain defects (102)