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[DOI] [PubMed] [Google Scholar] 81.Roman-Blas J.A., Contreras-Blasco M.A., Largo R., Álvarez-Soria M.A., Castañeda S., Herrero-Beaumont G
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Cardiac-specific deletion of mitochondrial complex I subunit NDUFS4 protein resulted in accumulation of NADH in mitochondria and elevation in NADH/NAD + in rodent heart, which correlated with the accelerated development of heart failure induced by pathological pressure overload or isoproterenol (42, 47)
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