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For instance, furosemide can inhibit metabolic pathways modulated by several enzymes including UDP-glucuronyltransferases (Sorgel et al., 1980), 11β-hydroxysteroid dehydrogenases (Escher et al., 1995
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Mitochondrial dysfunction is a common pathogenic feature of AD [176], evidenced by elevated ROS generation and lipid peroxidation, decreased mitochondrial membrane potential and altered mitochondrial morphology [176]
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