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Treatment of macrophages with oxPAPC led to a phenotypic switch of both M1 and M2 phenotypes into a new subtype named as Mox, characterized by exclusive upregulation of redox-regulated genes such as HO-1, sulforedoxin-1 (Srxn1), glutamate-cysteine ligase, modifier subunit (Gclm), glutamate-cysteine ligase, catalytic subunit (Gclc), thioredoxin reductase (Txnrd) 1, but also vascular endothelial growth factor (VEGF), nuclear receptor Nr4A2 (Nurr1) and Trb3 (Kadl et al., −/− mice had this phenotype

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