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N-Acetylcysteine in the Treatment of Acute Lung Injury: Perspectives and Limitations - PMC
It was also shown to induce apoptosis and cell cycle arrest in the G0/G1 phase, via the NLRC3/PI3K pathway, where it activates nucleotide oligomerization domain-like receptor subfamily C3 (NLRC3) and inhibits PI3K and cyclin D1-CDK4, leading to G0/G1 arrest [124]
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Autocrine factors that sustain glioma invasion and paracrine biology in the brain microenvironment
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