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The orexigenic agouti-related peptide and neuropeptide Y pathways and the anorexigenic pro-opiomelanocortin and cocaine-amphetamine-regulated transcript pathways of the ARC are well established, but more contemporary research has identified prolactin-releasing hormones and GCG neurons in the nucleus of the solitary tract as important anorexigenic pathways that respond to nutrient ingestion via vagus nerve stimulation [2]
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