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68 Meanwhile, the NF-κB pathway mediates inflammatory responses by upregulating chemokines like MCP-1 to recruit macrophages, and its activation in PSCs induced by TGF-β1 via p-TAK1 can further amplify the fibrotic microenvironment, forming an “inflammation-fibrosis” positive feedback loop

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144 Other promising PPAR agonists were evaluated in randomized controlled phase 2/3 trials of NASH patients including the dual PPARα/γ agonist Saroglitazar (NCT03061721) 145 and a pan-PPAR agonist Lanifibranor (NCT03008070)